About Us

Sovanni Cosmetics – Founder Bio

Sovanni was born in a lab, not a boardroom. I’m a cosmetic chemist with a chemistry degree and a master’s in biochemistry & molecular biology from UCLA, and I’ve spent years inside the pharmaceutical and beauty industries watching the same pattern repeat: shortcuts, fillers, synthetic overload, and products that look good on shelves but do nothing for skin health.

I wanted something better—formulas with intention, ingredients with integrity, and results that feel like skin coming home to itself. Sovanni is that answer.

At Sovanni, we formulate with mitochondrial-targeting antioxidants and cofactors that the rest of the skincare market has barely begun to explore. The mitochondria are the energy factories of every skin cell, producing the ATP that fuels repair, renewal, and resilience. When mitochondrial function declines—from UV exposure, stress, or simply age—skin loses its ability to bounce back. Most skincare stops at surface-level hydration or exfoliation. We go deeper, directly supporting the mitochondrial network with ingredients like ergothioneine, a super-antioxidant that selectively accumulates in mitochondria via its own dedicated transporter (OCTN-1) to neutralize ROS before oxidative damage cascades into visible aging. We pair it with coenzyme Q10, a lipid-soluble electron carrier essential to the mitochondrial respiratory chain that maintains ATP synthesis and reduces superoxide production. This combination doesn't just scavenge free radicals—it preserves mitochondrial membrane potential, protects mtDNA from mutation-driven deterioration, and sustains the energy gradient that healthy skin cells need to synthesize collagen and elastin. We also incorporate niacinamide, which serves as a precursor to NAD+, the critical coenzyme that fuels sirtuins—the longevity proteins that regulate cellular repair and stress resistance. Without adequate NAD+, the entire mitochondrial quality control system collapses. No one else is formulating with this level of deliberate mitochondrial science in a daily-wear makeup or skincare line. The result is cumulative: over weeks and months, skin doesn't just look better—it functions better, with improved firmness, reduced inflammation-driven aging, and a genuine radiance that comes from cells operating at full energy capacity. That is the Sovanni difference.

Sovanni is luxury with a backbone: science-led, nature-rooted, and created for people who are tired of trading their skin health for marketing claims. This brand is my rebellion against the shortcuts I’ve seen—and a promise that every jar, balm, and serum is built with the kind of quality I once wished existed.

Your skin deserves honesty. Sovanni makes it beautiful.

Sources:

  1. Ergothioneine Transporter and Mitochondrial Protection
    Safe and Effective Antioxidant: The Biological Mechanism and Potential Pathways of Ergothioneine in the Skin.
    Published in Molecules, 2023. Available via PMC, article ID PMC9967237.
    This review confirms that ergothioneine is transported into cells and mitochondria by the OCTN-1 transporter, accumulating where oxidative stress occurs to provide targeted antioxidant protection.

  2. Coenzyme Q10 and ATP Regeneration
    Schniertshauer, D., et al. Accelerated Regeneration of ATP Level after Irradiation in Human Skin Fibroblasts by Coenzyme Q10.
    Published in Photochemistry and Photobiology, 2016. Available via PubMed, PMID: 26946184.
    This study demonstrates that CoQ10 treatment preserves mitochondrial membrane potential and accelerates cellular ATP regeneration in skin fibroblasts after UV stress.

  3. NAD+ and Skin Cellular Bioenergetics
    Oblong, J.E. The Evolving Role of the NAD+/Nicotinamide Metabolome in Skin Homeostasis, Cellular Bioenergetics, and Aging.
    P&G Science Behind (OLAY). Available via INFONA.
    This paper establishes that cellular NAD+ pools are depleted in aged skin and that nicotinamide (niacinamide) restores these levels to support mitochondrial efficiency through sirtuin-dependent mechanisms.

  4. Mitochondrial Dysfunction and Skin Aging
    Patent published 2024.
    This patent details how mitochondrial dysfunction leads to ATP decline, excess ROS production, and accelerated skin aging, validating the role of mitochondrial cofactors in skincare.

  5. Mitochondrial Dynamics and UV-Induced Ferroptosis
    Icaritin alleviates UVB-induced skin damage by inhibiting ferroptosis via modulation of mitochondrial dynamics.
    Published in ScienceDirect, 2026. Open access.
    This research demonstrates that restoring mitochondrial function directly reduces skin cell death and collagen loss following UV exposure.